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78 Emerging studies demonstrate that there is a genetically programmed and regulated form of necrosis, termed necroptosis, that is dependent on receptor-interacting protein kinase 3 (RIPK3) and differs from apoptosis and necrosis

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36 Cell surface expression of MHCII and T-cell costimulatory molecules is markedly enhanced, whereas levels of inducible costimulator-ligand (ICOS-L), an inhibitory molecule involved in IL-10-dependent inhalational tolerance, 39 are decreased

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