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They act as coenzymes in mitochondrial energy metabolism by supporting enzymes in the Krebs cycle and in the electron transport chain, thus indirectly preventing excessive ROS formation, as they are not direct antioxidants because they do not chemically neutralise free radicals

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Compared with treatment with simvastatin alone, coadministration of simvastatin and anti-PD-L1 resulted in a notable decrease in tumor burden (Fig

These models provide complementary insights into the pathophysiology of AD, highlighting the distinct temporal and biochemical dynamics of amyloid pathology and cognitive decline