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HorikoshiNHwangSGatiCMatsuiTCastillo-OrellanaCRaubAGet alLong-range structural defects by pathogenic mutations in most severe glucose-6-phosphate dehydrogenase deficiency

Moreover, the preservation of glutathione levels, despite previously demonstrated hepatocellular injury, suggests that Tub-A is working via a metabolic pathway to prevent progression of liver injury after ischemia

Treatment with 0.1 mM GSH induced the expression of nodule development-related genes ( EFD and NF-YA1 ) in wild-type plants (Figure 10)

The TEM results were consistent with the changes in solution color and absorption spectra