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Abstract There is increasing awareness that oxidative stress may be implicated in the pathophysiology of autism spectrum disorder (ASD)

Clinically, biomarker-driven trials using liquid biopsies or exosome profiling could identify patient subgroups most likely to benefit from specific nanotherapies, optimizing therapeutic outcomes

Dissociated hippocampal neurons were transfected with mRFP- -actin and shMet RNA and receptor knock-down was allowed to take place for 48 hours prior to stimulating with 0.5 g (per well) HGF (10 ng/ml), Dihexa or Nle1-AngIV (both at 10 12 M)

Higher cAMP activates protein kinase A