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The pore-forming claudin (CLDN)-2 and barrier forming CLDN-12 seem to be crucial in the Ca 2+ transport through tight junction proteins (17)
Proposed pathways include modulation of multiple neurotransmitter systems (influencing serotonin, dopamine, norepinephrine, GABA, glutamate levels), possible involvement of NMDA receptors, alpha-1 adrenergic signaling (e.g., potentiating pineal acetyltransferase activity), inhibition of somatostatin release via dopaminergic routes, reduced ACTH/cortisol for stress buffering, and stimulated growth hormone or somatoliberin secretion
What is new, however, is the scale, accessibility, and uptake of faddish pharmaceuticals
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44 In the same line, AT 1 receptor-mediated vasoconstrictor effects of Ang IV were shown in rat renal interlobular arteries and in afferent and efferent arterioles of isolated perfused hydronephrotic kidneys, also at concentrations several fold higher than those required for Ang II
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