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Hepatic effects that are considered adverse include death, abnormal clinical chemistry parameters, obvious functional organ impairment, DNA and mitochondrial alteration, glutathione depletion, hepatocyte necrosis, enhanced replicative DNA synthesis, generation of reactive oxygen species, and increased peroxisome proliferation (in rodents) (Williams and Iatropoulos 2002)

Passioura, T

Oral supplements are available, but they may have lower bioavailability compared to IV administration

These models highlight the ovotoxic effects of environmental contaminants through oxidative pathways, providing frameworks for evaluating environmental risks and protective strategies ( A summary of the main animal models used to study ovarian oxidative stress and their key characteristics is presented in Table 1, providing an integrated overview of experimental strategies and their relevance in mimicking human reproductive pathologies