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Conversely, ectopic expression of constitutively active Rac1-Q61L led to significantly elevated FA numbers in control- or PFK-deficient cells (Fig

Our results thus suggest that Gclc is required to ensure the development of MZB, the mitochondrial ETC integrity in FoB, and the efficacy of antiviral humoral immunity

These findings underscore OS as a central mediator of -cell failure in diabetes, highlighting the need for targeted antioxidant strategies (see Figure 3) that mitigate ROS without disrupting physiological redox signaling (Zaher and Stephens, 2025)