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Early studies suggest potential benefits in conditions involving impaired intestinal motility or altered gut hormone signaling, including metabolic disorders and certain gastrointestinal diseases

In addition, endothelial dysfunction and initiation of atherogenesis are known to trigger by activating the nuclear factor-B (NF-B) signaling pathway and NLRP3 (NOD-like receptor protein 3 or NLR family pyrin domain containing 3) inflammasome pathway in endothelial cells both in vitro and in vivo (Seldin et al., 2016

Concentration-dependent cytotoxicity was observed as 5800 M gave a lower cell viability as compared to 580 M

The authors also deeply acknowledge the support from National Institute of Plant Genome Research and International Centre for Genetic Engineering and Biotechnology

Lundgren et al