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Therefore, dynamic analysis of redox changes during senescence evasion transitions is crucial to elucidate this phenomenon

We found that the GCL inhibitor BSO re-sensitized IRE1-null cells to erastin-induced ferroptosis, indicating that GCL is involved in IRE1-regulated GSH synthesis (Supplementary Fig

Calvarial bone regeneration by a combination of natural anorganic bovine-derived hydroxyapatite matrix coupled with a synthetic cell-binding peptide (PepGen): an experimental study in rats

In the PD brains, the loss in GSH in the SN results in high levels of 4HNE adducts [78]

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