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These proteins, acting as cyclin-dependent kinase (Cdk) inhibitors, lead to the arrest of the G1 phase, and activation of the senescence pathway [90]

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A 2020 study in Current Pharmaceutical Design confirmed that BPC 157 promotes the NO (nitric oxide) system, which plays a direct role in blood vessel formation and tissue protection

Hypoxia-induced stabilization of HIF1a leads to up-regulation of CXCL12, which in turn mediates recruitment of CXCR4-expressing endothelial progenitor cells from the bone marrow, thereby contributing to vasculogenesis [207]