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Modulation of Autoantibodies : Cellular Therapy and Stem Cells for Graves disease may influence the production and activity of autoantibodies, such as thyroid-stimulating immunoglobulins (TSIs), involved in stimulating thyroid hormone secretion

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Cell-derived soluble oligomers of human amyloid-beta peptides disturb cellular homeostasis and induce apoptosis in primary hippocampal neurons

Many studies have suggested that arrestins compete with G proteins for binding sites in the receptor transmembrane core and sterically preclude further G protein coupling, leading to receptor desensitization [1, 7]