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It also modulates retinal health, with studies showing normalized electroretinogram responses in aged rats (Bulletin of Experimental Biology and Medicine, 2002)

The clinical and biochemical features of diclofenac hepatotoxicity in humans and rats relates both to impairment of ATP synthesis by mitochondria, and to production of active metabolites, particularly N,5- dihydroxydiclofenac, which causes direct cytotoxicity

48 These possibilities remain speculative but are based on the complex network effects BPC-157 has in the body

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