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2011 May;3(5):464-78

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923 Genetic interference and pharmacological approaches targeting FADS1/2 make TNBC cells resistant to ferroptosis

Furthermore, CUL5-deficient macrophages exhibit a reduced activation threshold in response to IL-4, characterized by increased arginase 1 expression and a marked shift toward an anti-inflammatory phenotype, with upregulation of anti-inflammatory proteins such as Serpinb6a and Aldh5a1 (199)

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