[Abstract] Hepatol Int
In CKD, uremic toxin accumulation (e.g., indoxyl sulfate, acrolein), impaired antioxidant defense systems, and persistent inflammation collectively promote excessive production of reactive oxygen species (ROS), which disrupt erythrocyte membrane integrity, activate Ca 2+ -permeable cation channels, and trigger downstream eryptotic events (e.g., phosphatidylserine (PS) externalization, cell shrinkage, and cytoskeleton degradation) (Table 2 provides a detailed overview of their mechanisms, effects, and supporting evidence
Product Size:135g
Chit xut l hi: H tr dng m, lm du da, thc y ti to m v h tr phc hi cc vng da tn thng nh, gip da mm mi v hn ch kh rt
doi: 10.1136/fn.89.1.F57 206 TavesMDPlumbAWSandkamBAMaCvan der GugtenJGHolmesDTet al