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Research has demonstrated that the compound shows anti-proliferative activity in specific cancer cell lines, with HeLa cells showing concentration and time-dependent growth inhibition across a 0.1-500 M range while HEK-293 cells remain unaffected

Most observational studies examining vitamin C intake in relation to cancer incidence have either found no association or a modest, protective association

Bioavailability Depletion-repletion pharmacokinetic experiments demonstrated that plasma vitamin C concentration is tightly controlled by three primary mechanisms: intestinal absorption, tissue transport, and renal reabsorption (22)