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Previously, we have demonstrated significant decreases of plasma UA levels in either first-episode neuroleptic-nave patients with SZ (FENNS) patients (Reddy et al., 2003) or clinically stable patients with chronic SZ (Yao et al., 1998)

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The complexity, heterogeneity, and dynamic nature of cellular senescence highlight the need to develop more precise biomarkers for distinguishing pathological senescent cells from those exerting physiologically beneficial effects during aging

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