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Advances in the understanding of OA pathogenesis have facilitated the development of various disease-modifying osteoarthritis drugs (DMOADs), including small-molecule inhibitors targeting inflammatory and catabolic pathways, protein analogs promoting anabolic processes, and regenerative drugs modulating Wnt signaling to delay cartilage degeneration

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This result suggests GHK-Cu's effects on mitigating the deleterious effects of ROS [23, 60, 171, 172]

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