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KPV is used in inflammatory-signaling research

In mice, knockout of ChREBP has been shown to reduce hepatic fatty acid synthesis by 65% compared to wild-type controls, but also to promote insulin resistance, delayed glucose clearance, and severe intolerance to simple carbohydrates, such as sucrose and fructose (resulting in death in the majority of animals) due to an inability to shunt fructose into glycolytic pathways [48]

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Several mechanisms have been proposed for the development and progression of ALD including acetaldehyde toxicity, oxidative stress, increased intestinal permeability-induced endotoxemia, Kupffer cell activation, production of cytokines and chemokines, a compromised immune system, nutritional deficiencies, and altered methionine metabolism [33,52]