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Slc39a2-mediated zinc homeostasis modulates innate immune signaling in phenylephrine-induced cardiomyocyte hypertrophy

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Researchers considering extended protocols should be aware that this safety gap exists and should monitor for unexpected effects in tissues where NNMT has normal physiological functions

Researchers are particularly interested in its behavior under metabolic stress, where mitochondrial-derived peptides appear to play a regulatory role in how cells adapt energy production to changing demands