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This study aimed to investigate the effects of glutathione on gill tissue damage in juvenile grass carp under hypoxic stress and to explore its possible mechanisms of action in terms of tissue oxidative damage, apoptosis, autophagy, and endoplasmic reticulum stress

These trials included both single-dose pharmacokinetic studies and long-term efficacy trials extending up to 24 weeks of continuous administration

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activation of NF-B and the FABP4/NF-B feedback loop facilitates recruitment of immune cells to the CNS, amplifying inflammation [114, 115]

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