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Regional distribution of mutations of the ATP7B gene in patients with Wilson disease: impact on genetic testing

It can activate the production of metalloproteinases and anti-proteases that remove damaged proteins from the extracellular matrix macromolecules

Distinguishing direct pharmacological anti-inflammatory actions from secondary metabolic improvements requires further mechanistic studies and randomized controlled trials specifically designed to assess inflammatory outcomes as primary endpoints

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Background Selective delivery of chemotherapeutic agents to cancer cells to ensure the effectiveness of treatment while minimizing damage to healthy tissue is one of the most important aspects of cancer therapy [1,2,3]