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The Contribution of Homocysteine Metabolism Disruption to Endothelial Dysfunction: State-of-the-Art

Mechanistically, BPC-157 appears to modulate angiogenesis, upregulate growth factors such as VEGF and FGF, and influence nitric oxide pathways, contributing to enhanced vascularization, tissue repair, and anti-inflammatory effects

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Interestingly, incubation of human skeletal muscle cells with serum obtained from these subjects after the intervention resulted in an increase in PGC-1 expression, AMPK and SIRT1 activity, and mitochondrial biogenesis