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These claims have yet to be evaluated in humans and there is no research showing that it has this effect in test subjects

These processes play key roles to promote chemoresistance in tumors, where elevated GSH inactivates electrophilic chemotherapeutic agents (cisplatin, alkylators), GSTs enable drug conjugation, and MRP-catalyzed efflux reduces intracellular drug levels - while inhibiting therapy-induced oxidative stress at the same time

For example, it was demonstrated that GSH raised glutathione levels in the liver and plasma (Favilli et al ., 1997, Richie et al ., 2015)

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