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The consequences of thiamine deficiency such as increased oxidative stress, excitotoxicity, and inflammation, lead to cerebral vulnerability

This interaction with SIRT1, a key enzyme in cellular stress response and longevity, opens new research directions

Szczegln uwag powica si jego potencjalnemu udziaowi w mechanizmach zwizanych z telomerami, rytmem biologicznym, komunikacj komrkow oraz regulacj wybranych procesw molekularnych

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These findings are reported solely within research contexts and do not establish safety, efficacy, or therapeutic relevance