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Mark Ruscin J, Page RL, Scott J
Read the full article here: #NMRL #NeuropathicPain #PeripheralNeuropathy #SIRT3 #Mitochondria #FreeRadicalBiology #Neuroscience #Pharmacologyandtoxicology Department of Pharmacology and Toxicology, NIPER S.A.S
On one hand it is armed with robust superoxide dismutase (SOD), thioredoxin and glutathione redox system 4,5,6 , but on the other hand it lacks canonical unfolded protein response (UPR) components 7 , catalase and the classical glutathione peroxidase 8,9
4.7 Rheumatoid Arthritis (RA) RA is a highly prevalent disease with a complex, not yet fully understood etiology that is characterized by auto-Abs and excessive production of proinflammatory cytokines (including tumor necrosis factor-alpha, IL-1, IL-2, IL-6, IL-8, IL-17, interferon-gamma, receptor activator of NFB ligand, and granulocyte macrophage-colony stimulating factor)
