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The microbial metabolite trimethylamine N-oxide promotes antitumor immunity in triple-negative breast cancer

43 This suggests that this mtDNA mutation is a risk factor that requires interactions with nuclear mutations, polymorphisms or epigenetics and/or environmental triggers or modulators in order for the disease phenotype to be expressed

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This disparity raises a critical question for practitioners: How can one responsibly bridge the gap between compelling mechanistic promise and the current realities of the clinical evidence