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Given (i) that during fetal and early postnatal life, the brain is a fast growing organ, thus very high in energy and nutrient demand [10,11,12,13]

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IGF-1 therapy relieves hepatocyte premature senescence via inhibiting nuclear p53progerin interaction To further reveal the role of IGF-1 in oxidative stress-induced premature aging of hepatocytes, firstly, we explored whether short-term or prolonged exogenous IGF-1 treatment attenuated hepatocyte premature aging

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