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Abstract During human fasting, metabolic markers, including butyrates, carnitines, and branched-chain amino acids, are upregulated for energy substitution through gluconeogenesis and use of stored lipids

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Among them, NOD-like receptor (NLRP3) plays a role in inflammatory body formation and activation, as well as in the regulation of transforming growth factor beta-1 (TGF1), monocyte chemoattractant protein-1 (MCP-1), and tumor necrosis factor ligand superfamily member 2 (TNFSF2), all of which contribute to vascular complications in diabetes [65]

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