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Analysis of amylin consensus sequences suggests that human amylin is not optimized to minimize amyloid formation and provides clues to factors that modulate amyloidogenicity

doi: 10.1073/pnas.0504978102 86 LiT.ChiangJ

While each of these peptides has been studied individually in various in vitro and preclinical models , combining them is hypothesised to offer complementary modes of action

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