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It also induces the reverse transport of these transporters (Sulzer et al., 1995), leading to the release of monoamines from the neuronal cytoplasm into the synaptic cleft ( 2.1.2 Vesicular monoamine transporter 2 inhibition METH inhibits vesicular monoamine transporter 2 and disrupts monoamine storage in synaptic vesicles ( 2.1.3 Calcium dysregulation and excitatory neurotransmission METH enhances glutamate release, leading to NMDAR overactivation and increased calcium influx into neurons (Staszewski and Yamamoto, 2006)

doi:10.1016/j.ebiom.2025.105836 Compliance Statement This product is intended for laboratory research use only and is not approved for human or veterinary use

Damage to pre-oligodendrocytes and hypomyelination is unlikely to be solely due to autonomous cell-intrinsic vulnerability

We combine them with other strategies like personalized nutrition advice, hormone balancing, and prescription medications

5) Unlike pramlintide (short-acting amylin, FDA 2005) doesn't require 3-times daily dosing weekly format improves compliance