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Glutathione protects mitochondriafrom free radicals and the oxidative damage they cause
The expanded model shows astrocytic production of glutamine (pathway 1), its transfer to glutamatergic neurons (without indication of any extracellular space, because there is no other function for extracellular glutamine than astrocyte-to-neuron transfer) and extracellular release as the transmitter glutamate (pathway 2), and subsequent reuptake of glutamate and oxidative metabolism in astrocytes (pathway 3), with connections between pathways 1 and 3 shown as pathway 4
When applied topically, they are thought to interact with the skin in ways that support normal skin processes
Hence, it can be seen that many environmental pollutants are capable of inducing eryptosis in human RBCs, and that eryptosis may be an effective indicator for comparing the hematotoxicity of parent compounds and their metabolites [423], to assess the toxicity of their potential replacements [421, 422, 428], and to investigate the mechanisms of action of toxic metals [184, 420, 424, 435] and pesticides [436, 437]
Thyroid J 7 , 8487 (2018)
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