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Next, due to the poor bioavailability, permeability, and instability of both carnosine and quercetin 48 , their topical applications are limited

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Previous research has shown that excessive nitric oxide (NO) production, increased levels of oxidative stress, and cerebrovascular hypoperfusion are mayor pathomechanisms responsible for AD (Lepara et al

Oxidative stress, mitochondrial damage, ER stress, and inflammation converge on apoptotic signaling cascades

Stress can enhance fatigue when an individual performs a highly cognitive and attention-demanding task, e.g., long hours with saturated information [1]