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A.PihlajamkiJ.SoininenH.HiltunenM

The honest caveat is that this specific pairing has not been run through a published human combination trial, so the combined number remains a reasoned projection rather than a trial result

En los ltimos aos hemos presenciado un cambio radical en el tratamiento de las neoplasias slidas y hemticas

HGF/c-Met signaling is a well-established oncogenic pathway: c-Met is amplified or overexpressed in multiple cancers (gastric, lung, renal, hepatocellular) HGF/c-Met activation promotes tumor cell proliferation, survival, invasion, and metastasis c-Met inhibitors are FDA-approved cancer drugs (cabozantinib, crizotinib, capmatinib) A compound that potentiates HGF/c-Met signaling raises theoretical concerns about promoting tumor growth or transformation with chronic use

PMID: 7514608