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Glutathione perturbation in catalasedeficient cat2 is also linked to hypersensitive response (HR)-like lesions as part of a wide spectrum of defense responses that are conditionally induced in this line (Chaouch et al., 2010)

(2025), astrocytes gene signatures are upregulated during the early symptomatic stage, characterized by proliferation, and subsequently they decline as pro-inflammatory genes become upregulated during disease progression in TDP-43 mouse model

By the year 2000, specific gene suppression of glutaminase (GLS) became a promising strategy for effectively impeding tumor cell growth [13]

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