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doi: 10.1002/jmv.24810 131 LernerAMArizaMEWilliamsMJasonLBeqajSFitzgeraldJTet al

The introduction of wortmannin was found to significantly reverse the expression of PI3K and AKT, as well as the anti-apoptotic and anti-inflammatory effects of Dihexa, resulting in a decrease in the number of neuronal cells present in the cortex and the levels of IL-10, and an increase in the levels of TNF-alpha and IL-1-beta [1]

10% have intermediate enzyme activity, with an increased risk of toxicity that can be mitigated in most patients by dose reduction and careful follow-up

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