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If load strategy is absent, adding GHK-Cu may not meaningfully change outcomes

These picomolar levels of 15-HpETE-PE alone (Supplemental Figure 4A, generated under IL-13 plus arachidonic acid conditions) would be insufficient to directly oxidize the nanomolar changes caused by oxidation of GSH to GSSG observed in response to IL-13 (or the change with inhibition of 15LO1 or KD of ALOX15 /15LO1)

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Additionally, heterogeneous astrocytes distributed throughout the brain exacerbate disease progression and compromise the BBB by releasing inflammatory factors and chemokines [55, 56]