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The central objectives are to disrupt cancer cell metabolic plasticity, prevent interconversion between distinct metabolic states, and concurrently reprogram the immunometabolic landscape to restore therapeutic sensitivity [127]
The system focuses on treating symptoms instead of preventing them, missing the personalized care that could make all the difference
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Interestingly, glyoxalase activation under stress paralleled elevated expression of key artemisinin biosynthetic genes and higher in vivo artemisinin accumulation, suggesting a functional link between MG detoxification, redox regulation, and secondary metabolism during salinity stress in A
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