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Some studies point out the role of the bacteriophages and the virome in IBD pathogenesis, although this field has not been well studied yet (88, 89)

In human brain microvascular endothelial cells, downregulating hypoxia-inducible factor-1 activity and inhibiting its downstream gene vascular endothelial growth factor ameliorates the increased paracellular permeability and the alterations of distribution pattern of occludin and zonula occludens-1 induced by high glucose level [75]