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The study sought to clarify whether AOD-9604 produced its reported fat-reducing effects through the same receptor pathway as native growth hormone, and whether the 3-AR pathway was required for activity

Mice heterozygous for a disrupted UA oxidase transgene (UO X +/) exhibited diminished brain damage and enhanced functional recovery following ischemic stroke, prompted researchers to explore the potential role of UA treatment in animal stroke models [145]

doi: 10.21037/tau.2018.04.03

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