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NO is an endogenously produced vasodilator and is largely implicated in hemostatic mechanisms [39] while, in tail amputation and heparin-/warfarin-administration models, both the NO-synthase (NOS)-blocker, L-NAME (thought to have a prothrombotic effect) [3] and NOS-substrate, L-arginine [presented with an antithrombotic effect) [3] were little investigated

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A study showed that 500mg/day of vitamin C supplementation raised red blood cell glutathione levels by 47% in healthy adults

This was done by employing an engineered cyst(e)inase enzyme resulting in the suppression of tumor growth in breast and prostate cancer xenografts (53)

Crescenze IM, Gupta P, Adams G et al: Advanced management of patients with ulcerative interstitial cystitis/bladder pain syndrome