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I hERG ) amplitude (responsible for rapid delayed rectifier K + current)

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2 expression is highly upregulated by exogenous GSH supplementation indicating a substrate-dependent regulatory mechanism ( Given the growing body of literature on CHAC proteins, a comprehensive overview of all published work is beyond the scope of this review

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Xiao L, Xu X, Zhang F, Wang M, Xu Y, Tang D, Wang J, Qin Y, Liu Y, Tang C, He L, Greka A, Zhou Z, Liu F, Dong Z, Sun L (2017) The mitochondria-targeted antioxidant MitoQ ameliorated tubular injury mediated by mitophagy in diabetic kidney disease via Nrf2/PINK1