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targeting neutrophils to modify inflammation and improve host immunity
Neurologically, ALD/AMN can manifest with highly variable phenotypes, ranging from very aggressive and rapidly fatal inflammatory CNS demyelination in young boys to a more slowly progressive adult form either with cerebral white matter inflammatory lesions, or a more indolent progressive myelopathy and peripheral neuropathy (Moser, 1997)
-EC and GSH standards and UDE augmented BV-2 intracellular GSH content In our previous observations, we noted the anti-neuroinflammatory effect of a 4 h incubation with 100 M of -EC, GSH, or UDE containing 100 M -EC against the generation of proinflammatory cytokines induced by LPS
5 While effective interventions like iron supplementation, deworming, and malaria control are available for many direct causes of anaemia, new interventions must be developed and tested for other underlying causes
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