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Conclusive evidence from longitudinal clinical studies or targeted interventional trials in humans is still lacking

However, in OA, M2 numbers are limited and may shift toward an M1-like phenotype under sustained inflammatory stimuli, reducing their capacity to maintain iron homeostasis (109, 110).Together, these insights support the hypothesis that iron metabolism in M1 macrophages not only contributes to ferroptosis susceptibility in OA, but also represents a potential immunometabolic checkpoint for therapeutic intervention

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As expected, there were a total of eight mutants in five founders (including three females and two males) (Fig

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