GSH inhibits PSC activation induced by high glucose via the ROS/TGF/SMAD pathway in vitro The above results indicated an increase in pancreatic fibrosis in rats treated with LOsG, which was accompanied by an increase in ROS/TGF/SMAD signalling, and PSC proliferation and activation, which were all prevented by GSH
Organelle-specific sensors for monitoring Ca2+ dynamics in neurons
Although most of the ORFs corresponding to the TIGRFAMs present in the DIABLO networks were not taxonomically annotated, we could confirm some of the associations
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