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Hypothesising that decreased macrophage viability in an oxidative environment could be a contributing factor to the pathophysiology of CD, we found that monocyte-derived macrophages from individuals with active CD (but not those in clinical disease remission) have increased sensitivity to cell death induced by H 2 O 2

A novel protocol to provide a suitable cardiac model from induced pluripotent stem cells

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Mechanism of activation of mechanistic target of rapamycin complex 1 by methionine