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Physiologically, insulin signaling via the PI3K-Akt pathway enhances mitochondrial fatty acid oxidation by promoting the activity of carnitine palmitoyltransferase 1 (CPT1), which catalyzes the conjugation of long-chain acyl-CoA to carnitine for mitochondrial import [115,116,117]

During naive T-cell activation, exit from quiescence is supported by the uptake of specific extracellular amino acids

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