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During autophagy induction, global and promoter-specific H4K16ac levels, including those at ATG7 and ULK1 , decrease due to hMOF downregulation, indicating that reduced hMOF activity limits excessive autophagy and facilitates proper autophagic flux

The use of engineered vascular tissue, multiomic approaches, microphysiological systems, and computational modeling will provide insights into the underlying pathophysiology