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doi: 10.1002/jpen.2661 12

The CellTox reagent was added to the media in a 1:1,000 dilution, and the fluorescent signal was read using a FLUOstar Optima plate reader

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Furthermore, alanine mutations of the established glutathione-scaffold site residue D90 or of crucial residue K26 in monothiol PfGrx C32S/C88S slowed down the reaction and resulted in linear secondary diagrams for the first rate constant (Fig

These data suggest that while markers of fibrosis and autophagy are increased following liver-specific Gclc deletion, these are potentially due to the activation of NRF2