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In aggregate, a two-pronged paradigm is emerging: redesign cytotoxics to narrow their biodistribution, and concurrently deploy small-molecule fertoprotectants that intercept DNA damage, oxidative stress or apoptotic checkpoints specifically within the ovary

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Results Senescence-associated markers were enhanced in keloid fibroblasts and lesions Analysis of the heterogeneity of keloid cells revealed 28 cellular clusters (027) using unsupervised clustering of 34,043 cells based on established lineage-specific marker genes (Supplementary Fig