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Impact of ATXN3 Misfolding on the Subcellular Compartments: Mitochondria and ER Wild-type and mutant ATXN3 proteins have also been identified in the mitochondrial protein fraction of cells [138, 139], suggesting that mitochondrial function may be impaired as a result of ATXN3 misfolding and aggregation

Notably, pulsatile GH secretion persists even during continuous CJC-1295 stimulation, preserving physiological release patterns [2]

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